« Previous
Next »
Seizure: European Journal of Epilepsy
Volume 19, Issue 4
, Pages 226-231
, May 2010
GABAB receptor activation exacerbates spontaneous spike-and-wave discharges in DBA/2J mice
References
- . Proposal for revised classification of epilepsies and epileptic syndromes. Epilepsia. 1989;30:389–399
- . Typical absence seizures and their treatment. Arch Dis Child. 1999;81:351–355
- . Pharmacology of absence epilepsy. Trends Pharmacol Sci. 2003;24:542–549
- Activation of GABA(B) receptors reverses spontaneous gating deficits in juvenile DBA/2J mice. Psychopharmacology. 2007;194:361–369
- Beta and gamma range EEG power–spectrum correlation with spiking discharges in DBA/2J mice absence model: role of GABA receptors. Epilepsia. 2006;47:489–494
- . Increased thalamic inhibition in the absence seizure prone DBA/2J mouse. Epilepsia. 2008;49:921–925
- . Discriminative stimulus effects of flumazenil: perceptual masking by baclofen, and lack of substitution with gamma-hydroxybutyrate and its precursors 1,4-butanediol and gamma-butyrolactone. Behav Pharmacol. 2006;17:239–247
- . Cataleptic effects of gamma-hydroxybutyrate (GHB), its precursor gamma-butyrolactone (GBL), and GABAB receptor agonists in mice: differential antagonism by the GABAB receptor antagonist CGP35348. Psychopharmacology. 2007;192:407–414
- . Behavioral effects of gamma-hydroxybutyrate, its precursor gamma-butyrolactone, and GABA(B) receptor agonists: time course and differential antagonism by the GABA(B) receptor antagonist 3-aminopropyl(diethoxymethyl)phosphinic acid (CGP35348). J Pharmacol Exp Ther. 2009;330:876–883
- . Unravelling the brain targets of gamma-hydroxybutyric acid. Curr Opin Pharmacol. 2006;6:44–52
- . Enhancement of spike and wave discharges by GABAmimetic drugs in rats with spontaneous petit-mal-like epilepsy. Neurosci Lett. 1984;44:91–94
- . Improved pharmacological activity via pro-drug modification: comparative pharmacokinetics of sodium gammahydroxybutyrate and gamma-butyrolactone. Res Commun Chem Pathol Pharmacol. 1978;22:107–118
- . The gamma-hydroxybutyrate model of absence seizures: correlation of regional brain levels of gamma-hydroxybutyric acid and gamma-butyrolactone with spike wave discharges. Neuropharmacology. 1991;30:161–167
- . Gamma-Hydroxybutyric acid-induced seizures bear no relation to core temperature. Epilepsia. 1990;31:253–258
- . Determining the initial states in forward–backward filtering. IEEE Trans Signal Process. 1996;44:988–992
- . A procedure to estimate the fractal dimension of waveforms. Complexity Int. 1998;5
- . Spike–wave discharges and sleep spindles in rats. Acta Neurobiol Exp (Wars.). 1997;57:113–121
- . Genetic absence epilepsy in rats from Strasbourg—a review. J Neural Transm Suppl. 1992;35:37–69
- . Pharmacological profiles of generalized absence seizures in lethargic, stargazer and gamma-hydroxybutyrate treated model mice. Neurosci Res. 1997;29:17–22
- International Union of Pharmacology. XXXIII. Mammalian gamma-aminobutyric acid(B) receptors: structure and function. Pharmacol Rev. 2002;54:247–264
- . Characterization of the GABA autoreceptor in human neocortex as a pharmacological subtype of the GABA-B receptor. Eur J Pharmacol. 1994;263:311–314
- . Different subtypes of GABAB receptors are present at pre- and postsynaptic sites within the rat dorsolateral septal nucleus. J Neurophysiol. 1999;(June (81)):2875–2883
- Modification of GABA(B1) and GABA(B2) receptor subunits in the somatosensory cerebral cortex and thalamus of rats with absence seizures (GAERS). Epilepsy Res. 2003;55:39–51
- . Gamma-aminobutyric acid type B receptor splice variant proteins GBR1a and GBR1b are both associated with GBR2 in situ and display differential regional and subcellular distribution. J Biol Chem. 1999;274:27323–27330
- Pathway-specific action of gammahydroxybutyric acid in sensory thalamus and its relevance to absence seizures. J Neurosci. 2003;23:11469–11478
- . The distribution of gamma-hydroxybutyrate-induced Fos expression in rat brain: comparison with baclofen. Neuroscience. 2009;158:441–455
- . Neural networks of structures in which GABA-B receptors regulate absence seizures in the lethargic (lh/lh) mouse model. J Neurosci. 1995;15:7367–7376
- . The ontogeny of [3H]gamma-hydroxybutyrate and [3H]GABA-B binding sites: relation to the development of experimental absence seizures. Brain Res. 1994;659:147–156
- . Cataleptic effects of gamma-hydroxybutyrate (GHB) and baclofen in mice: mediation by GABA(B) receptors, but differential enhancement by N-methyl-d-aspartate (NMDA) receptor antagonists. Psychopharmacology. 2008;199:191–198
- . Responses to N-methyl-d-aspartate are enhanced in rats with petit mal-like seizures. J Neural Transm Suppl. 1992;35:97–108
- . Thalamic NMDA transmission in a genetic model of absence epilepsy in rats. Epilepsy Res. 1996;25:11–19
- . NMDA-NR1 and AMPA-GluR4 receptor subunit immunoreactivities in the absence epileptic WAG/Rij rat. Epilepsy Res. 2006;69:119–128
- Differential involvement of the dorsal hippocampus in passive avoidance in C57bl/6J and DBA/2J mice. Hippocampus. 2008;18:11–19
- High affinity binding sites for gamma-hydroxybutyric acid in rat brain. Life Sci. 1982;30:953–961
- . Gamma-hydroxybutyric acid binding sites in rat and human brain synaptosomal membranes. Biochem Pharmacol. 1984;15(33):2587–2590
- . Regulation of gamma-aminobutyric acid (GABA) release in cerebral cortex in the gamma-hydroxybutyric acid (GHB) model of absence seizures in rat. Neuropharmacology. 2000;39:427–439
- Dose-dependent absorption and elimination of gamma-hydroxybutyric acid in healthy volunteers. Eur J Clin Pharmacol. 1993;45:353–356
- Specific gamma-hydroxybutyrate-binding sites but loss of pharmacological effects of gamma-hydroxybutyrate in GABA(B)(1)-deficient mice. Eur J Neurosci. 2003;18:2722–2730
- Novel gamma-hydroxybutyric acid (GHB) analogs share some, but not all, of the behavioral effects of GHB and GABA-B receptor agonists. J Pharmacol Exp Ther. 2005;313:1314–1323
- . Antiabsence seizure activity of specific GABA-B and gamma-hydroxybutyric acid receptor antagonists. Pharmacol Biochem Behav. 1996;53:73–79
- . Opposite effects of GABAB receptor antagonists on absences and convulsive seizures. Eur J Pharmacol. 1997;332:245–255
- . Effects of the GABAB antagonist CGP 35348 on sleep–wake states, behaviour, and spike–wave discharges in old rats. Brain Res Bull. 1996;40:157–162
- The role of GABAB receptor activation in absence seizures of lethargic (lh/lh) mice. Science. 1992;257:398–401
- . Evidence for GABAB-mediated mechanisms in experimental generalized absence seizures. Eur J Pharmacol. 1992;31(213):343–349
- . GABAB receptor involvement in the control of genetic absence seizures in rats. Epilepsy Res Suppl. 1992;9:131–138
- . Involvement of intrathalamic GABAB neurotransmission in the control of absence seizures in the rat. Neuroscience. 1992;48:87–93
PII: S1059-1311(10)00049-X
doi: 10.1016/j.seizure.2010.02.007
© 2010 British Epilepsy Association. Published by Elsevier Inc. All rights reserved.
« Previous
Next »
Seizure: European Journal of Epilepsy
Volume 19, Issue 4
, Pages 226-231
, May 2010
